研究发现,与长期COVID神经症状有关的皮质素水平较低,这表明压力管理可能有助于控制症状。 Study finds low cortisol levels linked to neurological symptoms of Long COVID, suggesting stress management may help manage symptoms.
科罗拉多·博尔德大学的新研究表明,低皮质醇水平 -- -- 调节血压和睡眠觉循环的荷尔蒙 -- -- 可能是长期COVID神经症状的根源。 New research from the University of Colorado Boulder suggests that low cortisol levels, a hormone that regulates blood pressure and sleep-wake cycles, may underlie neurological symptoms of Long COVID. 这项发表在"大脑行为与免疫"杂志上的研究发现,长期COVID患者体内存在的免疫刺激蛋白质COVID-19抗原可以降低大脑中的皮质醇水平,刺激神经系统,并导致它对压力因素产生过度反应. The study published in the journal Brain Behavior and Immunity found that COVID-19 antigens, immune-stimulating proteins present in Long COVID patients, can lower cortisol levels in the brain, inflame the nervous system, and cause it to hyper-react to stressors. 研究人员发现,一种关键的抗炎荷尔蒙,皮质醇在长期的COVID病人经历的生理变化中起着重要作用。 Researchers discovered that cortisol, a crucial anti-inflammatory hormone, plays a significant role in the physiological changes experienced by long COVID patients. 调查结果表明,查明和尽量减少不同的应激因素可能有助于控制COVID长期症状,同时需要进一步研究以确定低分质醇如何助长这一状况。 The findings suggest that identifying and minimizing different stressors may help manage long COVID symptoms, while further research is needed to determine how low cortisol may contribute to the condition.