西北和UIC科学家发现了一种SARS-COV-2峰值蛋白质突变,使病毒进入中枢神经系统,这可能解释了长期的科氏神经症状。 Northwestern and UIC scientists identified a SARS-CoV-2 spike protein mutation enabling virus entry into the central nervous system, possibly explaining long Covid neurological symptoms.

西北大学和芝加哥伊利诺伊大学的科学家发现SARS-CoV-2的尖端蛋白中发生了突变,使病毒能够感染中枢神经系统,这可能解释了"长期Covid"的神经症状. Northwestern University and University of Illinois at Chicago scientists found a mutation in SARS-CoV-2's spike protein which allows the virus to infect the central nervous system, possibly explaining neurological symptoms of "long Covid." 这项发表在"自然微生物学"杂志上的研究揭示了蛋白中的几个变化,增加了病毒感染小鼠大脑的能力. The study published in Nature Microbiology revealed several alterations in the spike protein that increase the virus's ability to infect mice's brains. 这一发现可能导致有针对性的疗法,以保护大脑免受病毒的侵袭,并可能对治疗COVID-19病人的神经症状产生重大影响。 This discovery could lead to targeted therapies to protect the brain from the virus and may have significant implications for treating neurological symptoms in COVID-19 patients.