艾莫里大学的研究人员发现阿尔茨海默病患者大脑中的β粉样蛋白沉积物可以作为其他可能导致脑细胞损伤的蛋白质的支架. Emory University researchers find amyloid beta deposits in Alzheimer's patients' brains act as a scaffold for other proteins potentially causing brain cell damage.
艾默里大学研究人员质疑传统的阿尔茨海默氏理论,发现病人大脑中的氨基乙型氨基乙酰矿床是其他可能造成脑细胞损伤的蛋白质的脚手架。 Emory University researchers challenge traditional Alzheimer's theories, finding that amyloid beta deposits in patients' brains act as a scaffold for other proteins potentially causing brain cell damage. 在患有阿尔茨海默氏症和小鼠的人脑中，超过20种蛋白质与β淀粉样蛋白共同积累，这表明这些额外的蛋白质可能在脑损伤中发挥作用，从而为阿尔茨海默氏症和其他疾病带来潜在的新疗法和治疗靶点。 Over 20 proteins co-accumulate with amyloid beta in human brains with Alzheimer's and mice, suggesting these additional proteins may play a role in brain damage, leading to potential new therapies and treatment targets for Alzheimer's and other diseases.