康奈尔大学的研究人员发现了一种调节免疫反应引起的炎症的开关，涉及激酶 ITK 和钙。 Cornell researchers identified a switch regulating inflammation caused by immune responses, involving kinase ITK and calcium.
康奈尔大学的研究人员发现了一种调节免疫反应引起的炎症的开关。 Cornell researchers discovered a switch regulating inflammation caused by immune responses. 通过识别激酶 ITK（一种酶）和钙在控制这种转换中所起的作用，他们发现它决定了 T 细胞是发展为炎症 TH17 细胞还是抗炎 Treg 细胞。 By identifying the roles played by kinase ITK, an enzyme, and calcium in controlling this switch, they found that it determines whether T cells develop as inflammatory TH17 cells or anti-inflammatory Treg cells. 这一发现可能有助于临床医生控制炎症相关疾病，如自身免疫性疾病、心血管疾病和神经退行性疾病。 This finding could potentially help clinicians control inflammation-related conditions like autoimmune, cardiovascular, and neurodegenerative diseases.