一项研究将长期悲痛与大脑在奖赏和情感领域的改变联系起来,强调其生物学基础和需要更好的治疗。 A study links prolonged grief to brain changes in reward and emotion areas, highlighting its biological basis and need for better treatment.

一项新研究将影响约5%死者的长期悲痛症(PGD)与参与奖赏、动机和情绪处理的脑网络受到干扰联系起来。 A new study links prolonged grief disorder (PGD), affecting about 5% of bereaved people, to disrupted brain networks involved in reward, motivation, and emotion processing. 研究人员发现,在核丙苯和amygdala等区域,活动有所改变,这表明了持续渴望和情感痛苦的生物基础。 Researchers found altered activity in regions like the nucleus accumbens and amygdala, suggesting biological underpinnings for persistent yearning and emotional pain. 虽然PGD与抑郁症和创伤后精神紧张症有一些共同特点,但自2018年以来,PGD是截然不同和得到正式承认的。 Though PGD shares some features with depression and PTSD, it is distinct and formally recognized since 2018. 科学家们强调需要更好的诊断和治疗,呼吁进行更大规模的纵向研究,以确定大脑变化是造成还是造成长期悲伤。 Scientists stress the need for better diagnosis and treatment, calling for larger longitudinal studies to determine whether brain changes cause or result from prolonged grief.