一项研究显示,小鼠的粮食短缺重新规划免疫,促进快速反应细胞和节能。 Food shortages reprogram immunity in mice, boosting fast-acting cells and saving energy, a study shows.

食物短缺引发通过溶胶类、压力激素对免疫系统进行代谢性重新编程,这些激素将防御从能源密集的T型细胞转向反应更快的中子营养素,保存记忆T型细胞以获得长期免疫。 Food shortages trigger a metabolic reprogramming of the immune system via glucocorticoids, stress hormones that shift defense from energy-intensive T cells to faster-acting neutrophils, preserving memory T cells for long-term immunity. 在卡路里限制期间在小鼠身上观察到的这种适应有助于节能,同时通过将免疫细胞移到骨髓,保持抗感染性。 This adaptation, observed in mice during calorie restriction, helps conserve energy while maintaining infection resistance by relocating immune cells to the bone marrow. 根据2026年2月10日发表的一份研究报告得出的调查结果表明,免疫系统对养分短缺和其他压力因素进行了动态调整,对管理脆弱人群的感染和营养不良产生了影响。 The findings, based on a study published February 10, 2026, suggest the immune system dynamically adjusts to nutrient scarcity and other stressors, with implications for managing infections and malnutrition in vulnerable populations.