一项UQ研究发现,MIR-71在蠕虫体内保护mitocondria, 可能导致治疗老龄化、糖尿病和神经退化疾病。 A UQ study finds miR-71 protects mitochondria in worms, potentially leading to treatments for aging, diabetes, and neurodegenerative diseases.

昆士兰大学的科学家发现,称为MIR-71的微型RNA通过保护C. elegans蠕虫的mitochondria,防止有害压力信号的传播,有助于减少细胞压力。 Scientists at The University of Queensland discovered that a microRNA called miR-71 helps reduce cellular stress by protecting mitochondria in C. elegans worms, preventing harmful stress signals from spreading. 该研究发表在《自然通信》上,显示MIR-71调节了与老龄化、糖尿病和神经退化性疾病有关的应激反应。 The study, published in Nature Communications, shows miR-71 regulates stress responses linked to aging, diabetes, and neurodegenerative diseases. 研究人员说,在不开发抗老年药物的同时,研究人员表示理解这一机制可能会通过针对微型RNA来提高细胞抗御能力,从而导致新治疗新陈代谢和与年龄有关的疾病。 While not developing an anti-aging drug, researchers say understanding this mechanism could lead to new treatments for metabolic and age-related conditions by targeting microRNAs to improve cellular resilience.