杜克研究显示,从滑翔细胞转移到神经中可以减少小鼠和人体组织的慢性疼痛,为糖尿病和化疗引起的神经损伤提供可能的治疗方法。 Duke study shows healthy mitochondria transfer from glial cells to nerves reduces chronic pain in mice and human tissue, offering potential treatments for diabetic and chemo-induced nerve damage.

杜克大学的一项研究发现,通过从支撑性滑翔细胞转移到神经细胞,恢复神经细胞的米托孔德里亚,可减少小鼠和人体组织中的慢性神经疼痛,为糖尿病神经病和与化疗有关的神经损伤提供可能的治疗方法。 A Duke University study finds that restoring mitochondria to nerve cells via transfer from supporting glial cells reduces chronic nerve pain in mice and human tissue, offering potential treatments for diabetic neuropathy and chemotherapy-related nerve damage. 这一过程依赖于纳米管隧道和蛋白质MYO10, 能够改善能源流量,减少炎症,减轻疼痛长达48小时。 The process, dependent on tunneling nanotubes and the protein MYO10, improves energy flow and reduces inflammation, with pain relief lasting up to 48 hours. 捐赠细胞中的健康线粒体是有效的, 但受损的线粒体是不有效的. Healthy mitochondria from donor cells were effective, but damaged ones were not. 调查结果表明,加强这一自然修复机制可能导致神经疼痛新疗法。 The findings suggest that boosting this natural repair mechanism could lead to new therapies for nerve pain.