约翰·霍普金斯的研究将大脑蛋白质(CSE)与大鼠老年痴呆症的保护联系起来,指出一个新的治疗目标。 A Johns Hopkins study links a brain protein, CSE, to Alzheimer’s protection in mice, pointing to a new treatment target.

由约翰·霍普金斯(Johns Hopkins)领导的由NIH资助的研究发现,在脑中产生硫化氢的称为CSE的蛋白质可能是治疗阿尔茨海默氏病的有希望的目标。 A Johns Hopkins-led study funded by the NIH finds that a protein called CSE, which produces hydrogen sulfide in the brain, may be a promising target for Alzheimer’s treatments. 缺乏 CSE 的Mice 开发了记忆问题、神经发育减慢、血管损伤 — — 与阿尔茨海默氏病相关的特征 — — 暗示蛋白质有助于保护大脑健康。 Mice lacking CSE developed memory issues, reduced neuron growth, and blood vessel damage—features linked to Alzheimer’s—suggesting the protein helps protect brain health. 虽然人类应用仍然不确定,但调查结果突出表明了潜在治疗的新的生物途径。 While human applications remain uncertain, the findings highlight a new biological pathway for potential therapies.