新的研究将血管痴呆症与脑容器损伤、炎症和纳米塑料联系起来,这表明环境污染物可能发挥作用。 New research links vascular dementia to brain vessel damage, inflammation, and nanoplastics, suggesting environmental pollutants may play a role.

新的研究将血管痴呆症与包括炎症和废物清除受损在内的多种潜在脑容器问题联系起来,高级成像显示受影响个人脑组织中的纳米塑料。 New research links vascular dementia to multiple underlying brain vessel issues, including inflammation and impaired waste clearance, with advanced imaging revealing nanoplastics in brain tissue of affected individuals. 这些微粒的较高含量与病情恶化和发炎增加有关,表明环境污染物可能助长痴呆。 Higher levels of these particles correlate with worse disease and increased inflammation, suggesting environmental pollutants may contribute to dementia. 这项研究由新墨西哥大学Elaine Bearer博士领导,在《美国病理学期刊》上发表。 这项研究确定了阿尔茨海默氏病与阿尔茨海默氏病的重叠病理,包括氨基β,并提出了一个新的分类制度,以改进诊断。 The study, led by Dr. Elaine Bearer at the University of New Mexico and published in the American Journal of Pathology, identifies overlapping pathologies with Alzheimer’s, including amyloid beta, and proposes a new classification system to improve diagnosis. 在2 170万美元的NIH赠款的支持下,调查结果呼吁重新评估痴呆症原因,强调血管健康和环境暴露是关键因素。 Supported by a $21.7 million NIH grant, the findings call for reevaluating dementia causes, highlighting vascular health and environmental exposures as key factors.