研究发现,COVID-19可能因模仿人类蛋白质的病毒而引发自发免疫疾病。 Study finds COVID-19 may trigger autoimmune diseases due to virus mimicking human proteins.

最近对Immuno信息学的一项研究发现,COVID-19可能引发像1型糖尿病和多发性硬化症这样的自动免疫紊乱,其原因是分子模拟——病毒成分与人类蛋白质相似。 A recent study in ImmunoInformatics has found that COVID-19 may trigger autoimmune disorders like type 1 diabetes and multiple sclerosis due to molecular mimics—viral components that resemble human proteins. 研究人员利用先进的数据分析,查明了可导致免疫系统攻击人体的特定病毒蛋白质。 Using advanced data analysis, researchers identified specific viral proteins that could cause the immune system to attack the body. 研究表明,具有某些遗传特征的个人可能面临更高的风险,有可能导致对COVID引起的自免疫疾病采用新的治疗和预防方法。 The study suggests individuals with certain genetics might be at higher risk, potentially leading to new treatments and prevention methods for COVID-induced autoimmune diseases.