加州大学洛杉矶分校的研究将肥胖症和压力与通过蛋白质活化加快胰腺癌的生长联系起来。 UCLA study links obesity and stress to faster growth of pancreatic cancer via protein activation.

加州大学洛杉矶分校的研究人员发现,肥胖症和压力通过激活可促进癌症细胞生长的蛋白质CREB, 增加了突袭性胰腺癌的风险。 Researchers at UCLA found that obesity and stress increase the risk of aggressive pancreatic cancer by activating CREB, a protein that promotes cancer cell growth. 高脂肪饮食和压力下的小鼠，通常是通过社会孤立，更容易发展和迅速生长癌前病变。 Mice on high-fat diets and under stress, often through social isolation, were more likely to develop and rapidly grow precancerous lesions. 这项研究建议重新利用乙型阻塞剂等现有药物,以减轻这些癌症风险。 The study suggests repurposing existing medications like beta-blockers to mitigate these cancer risks.