研究者发现一些机制 帮助胰腺癌避免免疫系统, 建议新的治疗途径。 Researchers find mechanism helping pancreatic cancer evade immune system, suggesting new treatment paths.

来自台湾的研究人员确定了一种新机制,即TIMP1-CD63,它帮助胰腺癌细胞摆脱免疫系统并生长。 Researchers from Taiwan identified a new mechanism, TIMP1-CD63, which helps pancreatic cancer cells evade the immune system and grow. 这一机制加上DUSP2基因缺乏症,保护了大约90%的KRAS变异的癌症细胞,加速了癌症的蔓延。 This mechanism, along with a DUSP2 gene deficiency, protects about 90% of KRAS-mutated cancer cells, accelerating cancer progression. 该研究报告在《分子癌症》中发表,它表明,打破这一循环可能导致对胰腺癌的新治疗,这种疾病存活率不到10%。 The study, published in Molecular Cancer, suggests that disrupting this cycle could lead to new treatments for pancreatic cancer, a disease with a survival rate under 10%.