Vanderbilt大学医疗中心的研究将过度发烧与潜在的癌症风险联系起来,特别是通过TH1细胞线粒体压力和DNA损伤。 Study from Vanderbilt University Medical Center links excessive fever to potential cancer risk, particularly through Th1 cell mitochondrial stress and DNA damage.
Vanderbilt大学医疗中心的一项研究显示,发烧可增强免疫细胞活动,但也在某些T细胞,特别是T1细胞中诱发米托分体压力和DNA损害。 A study from Vanderbilt University Medical Center reveals that fever can enhance immune cell activity but also induces mitochondrial stress and DNA damage in certain T cells, particularly Th1 cells. 研究在《科学免疫学》中发表,研究表明,虽然中度发热是有益的,但过热可能助长与慢性炎症有关的癌症发育,而慢性炎症约占癌症病例的25%。 Published in Science Immunology, the research suggests that while moderate fever is beneficial, excessive heat may contribute to cancer development linked to chronic inflammation, which accounts for about 25% of cancer cases. 需要进一步研究,以证实这些调查结果。 Further studies are needed to confirm these findings.